RT Book, Section
A1 Arbustini, Eloisa
A1 Maron, Barry J.
A1 Narula, Navneet
A1 Narula, Jagat
A2 Fuster, Valentin
A2 Narula, Jagat
A2 Vaishnava, Prashant
A2 Leon, Martin B.
A2 Callans, David J.
A2 Rumsfeld, John S.
A2 Poppas, Athena
SR Print(0)
ID 1202447930
T1 Classification of Cardiomyopathies
T2 Fuster and Hurst's The Heart, 15e
YR 2022
FD 2022
PB McGraw-Hill Education
PP New York, NY
SN 9781264257560
LK accesscardiology.mhmedical.com/content.aspx?aid=1202447930
RD 2024/04/23
AB Chapter  SummaryThis  chapter  reviews  the  evolving  classification  of  cardiomyopathies  over  the  last  65  years,  which  were  first  defined  in  1957  as  noncoronary  cardiomyopathies  or  isolated  noncoronary  myocardial  diseases  (see  Fuster  and  Hurst’s  Central  Illustration).  In  2013,  the  World  Heart  Federation  (WHF)  endorsed  a  systematic  classification  referred  to  as  MOGE(S),  which  combined  two  complementary  concepts  from  the  previous  American  Heart  Association  (AHA)  and  European  Society  of  Cardiology  (ESC)  recommendations.  The  system  of  classification  describes  morphofunctional  presentation  (M),  organ  involvement  (O),  genetic  basis  (G),  precise  description  of  (genetic  or  acquired)  etiology  (E)  and  functional  New  York  Heart  Association  (NYHA)  class  and  American  College  of  Cardiology  (ACC)–AHA  status  (S).  In  the  2019  classification  of  cardiomyopathies  in  children,  the  AHA  incorporated  the  elements  of  MOGE(S)  in  a  hierarchy  system  based  on  the  structural  and  functional  phenotype  with  genetic  and  nongenetic  causes  as  subcategories.  Going  forward,  each  new  formula  for  reporting  the  classification  will  have  to  realize  the  need  for  short  and  rapid  communication  tools  that  can  be  universally  adopted,  but  we  must  also  recognize  that  the  acceptance  of  every  new  proposal  by  the  clinical  community  takes  time.  Any  commitment  of  the  clinical  community  has  to  go  beyond  the  phenotype  and  include  the  cause  as  mandatory  information,  so  that  all  cardiologists  share  a  common  diagnostic  language.