RT Book, Section A1 Spragg, David A1 Calkins, Hugh A2 Fuster, Valentin A2 Narula, Jagat A2 Vaishnava, Prashant A2 Leon, Martin B. A2 Callans, David J. A2 Rumsfeld, John S. A2 Poppas, Athena SR Print(0) ID 1202446998 T1 Supraventricular Tachycardia: Atrial Tachycardia, Atrioventricular Nodal Reentry, and Wolff-Parkinson-White Syndrome T2 Fuster and Hurst's The Heart, 15e YR 2022 FD 2022 PB McGraw-Hill Education PP New York, NY SN 9781264257560 LK accesscardiology.mhmedical.com/content.aspx?aid=1202446998 RD 2024/10/10 AB Chapter SummaryThis chapter discusses the mechanisms and treatment of supraventricular tachycardia (SVT), a term that encompasses three distinct arrhythmias: atrioventricular nodal reentrant tachycardia (AVNRT), atrioventricular reciprocating tachycardia (AVRT), and atrial tachycardia (AT). These arrhythmias, while not life threatening, can cause significant symptoms in afflicted patients including palpitations, dizziness, and syncope. AVNRT accounts for roughly two-thirds and AVRT for nearly one-third of SVT cases. AVNRT and AVRT are reentrant rhythms, while AT can be due to triggering or automaticity (see Fuster and Hurst’s Central Illustration). The reentrant circuit for AVNRT includes pathways with distinct electrophysiological properties within the atrioventricular node, while the circuit for AVRT includes the atrioventricular node and an extranodal accessory pathway. Frequently, SVTs can be terminated acutely with vagal maneuvers. Long-term management options include chronic suppression with antiarrhythmic medications or definitive therapy with catheter ablation.