RT Book, Section A1 Calkins, Hugh A2 Fuster, Valentin A2 Harrington, Robert A. A2 Narula, Jagat A2 Eapen, Zubin J. SR Print(0) ID 1161718472 T1 SUPRAVENTRICULAR TACHYCARDIA: ATRIAL TACHYCARDIA, ATRIOVENTRICULAR NODAL REENTRY, AND WOLFF-PARKINSON-WHITE SYNDROME T2 Hurst's The Heart, 14e YR 2017 FD 2017 PB McGraw-Hill Education PP New York, NY SN 9780071843249 LK accesscardiology.mhmedical.com/content.aspx?aid=1161718472 RD 2024/04/19 AB Supraventricular tachycardias (SVTs) include all tachyarrhythmias that either originate from or incorporate supraventricular tissue in a reentrant circuit. The ventricular rate may be the same or less than the atrial rate, depending on the atrioventricular (AV) nodal conduction. The term paroxysmal supraventricular tachycardia (PSVT) refers to a clinical syndrome characterized by a rapid, regular tachycardia with an abrupt onset and termination. Approximately two-thirds of cases of PSVT result from AV nodal reentrant tachycardia (AVNRT). Orthodromic AV reentrant tachycardia (AVRT), which involves an accessory pathway (AP), is the second most common cause of PSVT, accounting for approximately one-third of cases. The term Wolff-Parkinson-White (WPW) syndrome designates a condition comprising both preexcitation and tachyarrhythmias. Atrial tachycardias, which arise exclusively from atrial tissue, account for approximately 5% of all cases of PSVT.1 The purpose of this chapter is to review the mechanism, clinical features, and approach to diagnosis and treatment of patients with AVNRT and AP-mediated tachycardias (including WPW syndrome). Particular attention is focused on reviewing management guidelines developed by the American Heart Association (AHA), American College of Cardiology (ACC), and Heart Rhythm Society (HRS; formerly known as the North American Society of Pacing and Electrophysiology).1 We will also provide a brief overview of atrial tachycardia.