RT Book, Section
A1 Ferrara, Angela
A1 Adjedj, Julien
A1 Bruyne, Bernard De
A2 Samady, Habib
A2 Fearon, William F.
A2 Yeung, Alan C.
A2 King III, Spencer B.
SR Print(0)
ID 1146595261
T1 Hemodynamic Assessment of Renal Artery Stenosis
T2 Interventional Cardiology, 2e
YR 2017
FD 2017
PB McGraw-Hill Education
PP New York, NY
SN 9780071820363
LK accesscardiology.mhmedical.com/content.aspx?aid=1146595261
RD 2024/04/20
AB Renal  artery  stenosis  (RAS)  is  the  main  cause  of  “secondary”  arterial  hypertension.  RAS  is  found  in  0.5%  to  5%  of  hypertensive  patients.  RAS  is  most  often  atherosclerotic  in  nature  and  less  frequent  due  to  fibromuscular  hyperplasia.1  The  prevalence  of  RAS  is  approximately  2%  in  unselected  patients  but  reaches  40%  in  older  patients  or  in  patients  with  multiple  risk  factors  for  atherosclerosis  or  with  documented  atherosclerosis  in  other  vascular  territories.2,3  Due  to  improvements  in  vascular  imaging  by  ultrasound,  magnetic  resonance  imaging,  computed  tomography,  and  angiography  and  due  to  the  larger  proportion  of  elderly  patients  undergoing  coronary  angiography,  the  finding  of  a  RAS  on  angiography  is  a  frequent  occurrence.  “Drive-by”  renal  angiograms  are  frequently  performed  during  coronary  angiography  procedures.  However,  as  is  the  case  in  the  coronary  arteries,  the  relationship  between  anatomic  findings  and  the  functional  repercussion  of  a  given  stenosis  is  poor.4