TY - CHAP M1 - Book, Section TI - Ventricular Tachycardia A1 - Prystowsky, Eric N. A2 - Prystowsky, Eric N. A2 - Klein, George J. A2 - Daubert, James P. Y1 - 2020 N1 - T2 - Cardiac Arrhythmias: Interpretation, Diagnosis, and Treatment, 2e AB - Ventricular tachycardia (VT) is protean in form, duration, clinical setting, and prognosis. There is no uniformly accepted classification of VT, and this arrhythmia may be subdivided using many factors, as noted in Table 8-1. Unfortunately, no single subcategory provides enough information for correct classification of all patients with VT, and in many instances there is substantial crossover. For example, in many patients the mechanism of tachycardia is not certain. Whereas reentry is the presumptive mechanism for sustained VT in patients who have coronary artery disease with a previous myocardial infarction and ventricular scar, the mechanism of exercise-induced VT may include enhanced automaticity, triggered activity, and probably reentry. Thus, our present state of knowledge regarding mechanism of VT precludes use of this sole criterion for classification. The other potential classification variables listed in Table 8-1 also have deficiencies. It is often more useful to define a set of variables for a particular type of tachycardia so as to provide the clinician with a meaningful approach to the patient who has that arrhythmia. For example, patients with no definable structural heart disease who have right bundle branch block, left axis morphology sustained VT are a select subgroup for whom the location of this tachycardia is known and response to therapy—for example, endocardial radiofrequency catheter ablation or oral verapamil—has been well-documented.1-3 We will approach the classification of VT by clinical presentation in order to aid the clinician taking care of these patients. SN - PB - McGraw Hill CY - New York, NY Y2 - 2024/04/20 UR - accesscardiology.mhmedical.com/content.aspx?aid=1176522444 ER -